Search results for " Neuropeptide Y"

showing 7 items of 7 documents

Effects of nifedipine on renal and cardiovascular responses to neuropeptide y in anesthetized rats

2021

Neuropeptide Y (NPY) acts via multiple receptor subtypes termed Y1, Y2 and Y5. While Y1 receptor-mediated effects, e.g., in the vasculature, are often sensitive to inhibitors of L-type Ca2+ channels such as nifedipine, little is known about the role of such channels in Y5-mediated effects such as diuresis and natriuresis. Therefore, we explored whether nifedipine affects NPY-induced diuresis and natriuresis. After pre-treatment with nifedipine or vehicle, anesthetized rats received infusions or bolus injections of NPY. Infusion NPY (1 µg/kg/min) increased diuresis and natriuresis, and this was attenuated by intraperitoneal injection of nifedipine (3 µg/kg). Concomitant decreases in heart ra…

0301 basic medicineMaleReceptors Neuropeptidemedicine.medical_treatmentMedizinPharmaceutical ScienceOrganic chemistry030204 cardiovascular system & hematologyAnalytical ChemistryReceptors G-Protein-CoupledY<sub>1</sub> receptor0302 clinical medicineBolus (medicine)QD241-441Drug DiscoveryMedicineY1 receptorblood pressureNeuropeptide Y receptorCalcium Channel Blockershumanitiesnifedipinemedicine.anatomical_structureChemistry (miscellaneous)Molecular MedicineY5 receptormedicine.drugmedicine.medical_specialtyneuropeptide YIntraperitoneal injectionnatriuresisDiuresisArticleNatriuresis03 medical and health sciencesY<sub>5</sub> receptorNifedipineInternal medicinemental disordersAnimalsPhysical and Theoretical ChemistryRats Wistarbusiness.industryrenal blood flowRatsReceptors Neuropeptide Ydiuresis030104 developmental biologyEndocrinologyRenal blood flowVascular resistancebusiness
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Evidence for hypothalamic ketone bodies sensing: impact on food intake and peripheral metabolic responses in mice

2016

Monocarboxylates have been implicated in the control of energy homeostasis. Among them, the putative role of ketone bodies produced notably during high-fat diet (HFD) has not been thoroughly explored. In this study, we aimed to determine the impact of a specific rise in cerebral ketone bodies on food intake and energy homeostasis regulation. A carotid infusion of ketone bodies was performed on mice to stimulate sensitive brain areas for 6 or 12 h. At each time point, food intake and different markers of energy homeostasis were analyzed to reveal the consequences of cerebral increase in ketone body level detection. First, an increase in food intake appeared over a 12-h period of brain keton…

Blood GlucoseMale0301 basic medicineobesitynervous-systemPhysiology[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionEndocrinology Diabetes and MetabolismKetone BodiesEnergy homeostasisEatingMicebodiesHomeostasisGlucose homeostasisoxidative stressAgouti-Related ProteinNeuropeptide YPhosphorylationmonocarboxylate transporters2. Zero hunger[ SDV.MHEP.PHY ] Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]fat massHypothalamusKetone bodiesStarvation responseketogenic mediterranean dietweight-lossmedicine.medical_specialtybeta-hydroxybutyrateHypothalamusBiologyDiet High-Fat03 medical and health sciencesInsulin resistancerat-brainPhysiology (medical)Internal medicinemedicine[SDV.MHEP.PHY]Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]Animalsglucose homeostasisAdenylate Kinase/metabolism; Agouti-Related Protein/metabolism; Animals; Blood Glucose; Diet High-Fat; Eating/drug effects; Eating/physiology; Energy Metabolism/drug effects; Energy Metabolism/physiology; Gluconeogenesis/drug effects; Gluconeogenesis/physiology; Homeostasis; Hypothalamus/drug effects; Hypothalamus/metabolism; Insulin Resistance/physiology; Ketone Bodies/pharmacology; Male; Mice; Mice Inbred C57BL; Neuropeptide Y/metabolism; Phosphorylation/drug effectsenergy homeostasisAdenylate KinaseGluconeogenesismedicine.diseaseMice Inbred C57BL030104 developmental biologyEndocrinologyGluconeogenesislow-carbohydrateInsulin ResistanceEnergy Metabolism[SDV.AEN]Life Sciences [q-bio]/Food and NutritionHomeostasis
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Pancreatic polypeptide stimulates mouse gastric motor activity through peripheral neural mechanisms

2016

Background Pancreatic polypeptide (PP) is supposed to be one of the major endogenous agonists of the neuropeptide Y4 receptor. Pancreatic polypeptide can influence gastrointestinal motility, acting mainly through vagal mechanisms, but whether PP acts directly on the stomach has not been explored yet. The aims of this study were to investigate the effects of PP on mouse gastric emptying, on spontaneous tone of whole stomach in vitro and to examine the mechanism of action. Methods Gastric emptying was measured by red phenol method after i.p. PP administration (1–3 nmol per mouse). Responses induced by PP (1–300 mmol L−1) on gastric endoluminal pressure were analyzed in vitro in the presence o…

Male0301 basic medicinemedicine.medical_specialtyPhysiologyGastric emptyingMuscarinic AntagonistsBiologyEndocrine and Autonomic SystemMice03 medical and health sciencesOrgan Culture Techniques0302 clinical medicineInternal medicineMuscarinic acetylcholine receptormedicineAnimalsPancreatic polypeptidePeripheral NervesPancreatic polypeptideReceptorAntrumDose-Response Relationship DrugGastric emptyingEndocrine and Autonomic SystemsStomachGastroenterologyMotilityAcetylcholineReceptors Neuropeptide YMice Inbred C57BL030104 developmental biologyEndocrinologymedicine.anatomical_structureMechanism of actionTachykininmedicine.symptomEnteric nervous systemGastrointestinal Motility030217 neurology & neurosurgeryAcetylcholinemedicine.drug
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Pharmacogenomics of cantharidin in tumor cells.

2014

Cantharis vesicatoria (blister beetle) is used in Chinese medicine and has been categorized as highly toxic in the Chinese pharmacopeia. In Europe, Cantharis patches have been used since ages to treat various skin-related diseases. We investigated the cytotoxicity of the Cantharis ingredient, cantharidin, in 41 tumor cell lines (Oncotest panel) and compared the results with those of 60 cell lines of the National Cancer Institute, USA. We found profound activity at low micromolar concentrations (log ₁₀IC₅₀ values between -6.980 and 5.009 M). Cantharidin bound to protein phosphatase 2A (PP2A) with higher affinity (-8.12 kcal/mol) than to PP1 (-6.25 kcal/mol) in molecular docking analyses. Usi…

Models MolecularProtein ConformationBlister beetleBiologyCantharisBiochemistryFas ligandGene Expression Regulation Enzymologicchemistry.chemical_compoundCell Line TumorGene expressionAnimalsHumansRNA MessengerCytotoxicityOligonucleotide Array Sequence AnalysisPharmacologyCantharidinBinding SitesMolecular Structurebiology.organism_classificationMolecular biologyReceptors Neuropeptide YchemistryCell cultureApoptosisPharmacogeneticsImmunologyCantharidinBiochemical pharmacology
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Origin of the prolactin-releasing hormone (PRLH) receptors: evidence of coevolution between PRLH and a redundant neuropeptide Y receptor during verte…

2004

We present seven new vertebrate homologs of the prolactin-releasing hormone receptor (PRLHR) and show that these are found as two separate subtypes, PRLHR1 and PRLHR2. Analysis of a number of vertebrate sequences using phylogeny, pharmacology, and paralogon analysis indicates that the PRLHRs are likely to share a common ancestry with the neuropeptide Y (NPY) receptors. Moreover, a micromolar level of NPY was able to bind and inhibit completely the PRLH-evoked response in PRLHR1-expressing cells. We suggest that an ancestral PRLH peptide started coevolving with a redundant NPY binding receptor, which then became PRLHR, approximately 500 million years ago. The PRLHR1 subtype was shown to have…

Prolactin-releasing hormoneGeneticsBase SequenceMolecular Sequence DataBiologyNeuropeptide Y receptorProlactinReceptors G-Protein-CoupledReceptors Neuropeptide YEvolution MolecularPhylogeneticsMolecular evolutionHormone receptorGene DuplicationGene duplicationVertebratesGeneticsAnimalsHumansReceptorPhylogenyGenomics
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Receptor-mediated uptake of boron-rich neuropeptide y analogues for boron neutron capture therapy.

2014

Peptidic ligands selectively targeting distinct G protein-coupled receptors that are highly expressed in tumor tissue represent a promising approach in drug delivery. Receptor-preferring analogues of neuropeptide Y (NPY) bind and activate the human Y1 receptor subtype (hY1 receptor), which is found in 90% of breast cancer tissue and in all breast-cancer-derived metastases. Herein, novel highly boron-loaded Y1 -receptor-preferring peptide analogues are described as smart shuttle systems for carbaboranes as (10) B-containing moieties. Various positions in the peptide were screened for their susceptibility to carbaborane modification, and the most promising positions were chosen to create a mu…

inorganic chemicalsStereochemistrymedia_common.quotation_subjectMolecular Sequence Datachemistry.chemical_elementPeptideBoron Neutron Capture TherapyBreast NeoplasmsBiochemistrySolid-phase synthesisDrug DiscoveryChlorocebus aethiopsAnimalsHumansNeuropeptide YAmino Acid SequenceGeneral Pharmacology Toxicology and PharmaceuticsReceptorInternalizationBoronBoranesmedia_commonPharmacologychemistry.chemical_classificationChemistryOrganic ChemistryReceptor-mediated endocytosisNeuropeptide Y receptorReceptors Neuropeptide YHEK293 CellsDrug deliveryCOS CellsMolecular MedicineFemaleChemMedChem
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Effect of Acetaldehyde Intoxication and Withdrawal on NPY Expression: Focus on Endocannabinoidergic System Involvement

2014

Acetaldehyde (ACD), the first alcohol metabolite, plays a pivotal role in the rewarding, motivational and addictive properties of the parental compound. Many studies have investigated the role of ACD in mediating neurochemical and behavioral effects induced by alcohol administration, but very little is known about the modulation of neuropeptide systems following ACD intoxication and withdrawal. Indeed the neuropeptide Y (NPY) system is altered during alcohol withdrawal in key regions for cerebrocortical excitability and neuroplasticity. The primary goal of this research was to investigate the effects of ACD intoxication and withdrawal by recording rat behavior and by measuring neuropeptide …

medicine.medical_specialtylcsh:RC435-571hippocampusnucleus accumbensHippocampusNeuropeptidePhysical dependenceNucleus accumbensendocannabinoidergic systemNeurochemicallcsh:PsychiatryInternal medicinemental disordersmedicineendocannabinoid systemneuropeptide Y expressionOriginal ResearchPsychiatryacetaldehyde withdrawal neuropeptide Y expression endocannabinoidergic system hippocampus nucleus accumbensKindlingAlcohol dependenceacetaldehyde withdrawal neuropeptide Y expression endocannabinoidergic system hippocampusnucleus accumbensNeuropeptide Y receptorPsychiatry and Mental healthEndocrinologyacetaldehyde withdrawalmedicine.symptomPsychologyFrontiers in Psychiatry
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